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Generally speaking, skin cancers are divided into two categories: melanoma and non-melanoma skin cancers. Skin cancer tends to start in the outer layer of the skin where it is visible to the naked eye, making early diagnosis a little easier than other forms of cancer.
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Any cell can be transformed into a cancer cell—and there are many, many ways this can happen depending on the specific cell. This is just one of the reasons there may never be a single “cure” for cancer in general; cancer represents well over 100 different types. Each type of cancer may have different mechanisms contributing to its transformation and each may have different points at which it can be treated.
One of the obstacles in catching skin cancer early is that normal skin blemishes, such as moles, are often overlooked for long periods of time as being nothing to worry about. To help distinguish everyday moles from possible melanomas, use the mnemonic “ABCDE.”
Types of Skin Cancer
Most skin cancers—with the exception of melanomas—do not spread very quickly, if at all, and many can be successfully treated with surgical procedure called excision.
In mice, for example, cannabinoids have been shown to reduce the size of breast tumors and lower the frequency of metastasis. Many other studies have indicated that the endocannabinoid system is functional in the skin and may play a role in skin cancers, especially inflammatory skin cancers.
Underlying all cancers is the regulation of gene expression. For every type of cancer, there is a different gene (or genes) that becomes activated or deactivated causing the cell to become cancerous.
CBD oil has nearly no toxicity and no major side effects 8 , meaning patients shouldn’t worry about over-dosing. In fact – if a patient has positive results using the oil, it can be worth trying to increase the dosage to accelerate the rate of recovery.
Skin cancer is one of the most common, most treatable, and yet also one of the deadliest types of cancer. It typically presents as a skin lesion that won’t heal and tends to keep growing and ulcerating indefinitely. When caught at an early stage, this cancer can often be cured fairly easily by removing the affected area. Things can get a bit trickier once it has metastasized – which means it has progressed below the surface of the skin and burrowed into vital organs.
Anyone who wishes to catch up to the medical research around cannabis oil as a possible cancer treatment will find massive amounts of information available online. At this point in history, cannabis has been extensively studied 3 with promising results that already led to the development of the first cannabinoid-based pharmaceuticals.
CBD dosage for skin cancer
In recent years, there has been increasing attention to the use of cannabis and CBD oil for skin cancer 1 .
It’s worth noting the results seem to be dependent on the dose, and it typically takes a strong concentration of cannabis oil that’s applied regularly and in sufficient quantity to a skin cancer lesion, in order to effectively heal it. As well as applying the CBD oil on the cancerous lesion (if possible), it’s recommended to also consume CBD oil internally to boost the immune system 7 as well as to minimize the side effects and improve the success rate of conventional cancer treatments.
Even though it was only discovered in recent decades, this is the largest neurotransmitter system in the human body; it seems to have a key role in regulating the maintenance of physical and mental health from a cellular level, and it’s activated by the unique compounds available in the cannabis plant. These compounds, including CBD, THC and several other cannabinoids effectively seem to mimic substances that our body produces naturally, which are extremely important to the maintenance of good health 4 .
There is plenty of medical research exploring and evaluating the relationship between CBD and cancer in general, which is giving hope to patients suffering specifically form skin cancer. Studies have shown that cannabidiol can promote the apoptotic death of cancer cells 5 acting independently of the cb1 and cb2 receptors. The way this happens remains not fully known, for now, however it is assumed that it is due to the compound’s ability to enhance the production of reactive oxygen species in cancer cells 6 .